Heart attacks could be predicted YEARS in advance as research shows that people with high levels of calcium in the aorta are up to four times more likely to be precipitated
- A new study suggests that heart attacks can be predicted from a simple X-ray
- High levels of calcium in the aorta can increase the risk of a heart attack
- Factors that stimulate arteriosclerosis include poor nutrition, smoking and genetics
Heart attacks could be predicted years in advance – via a simple X-ray, according to new research.
People with high levels of calcium in the aorta – an important blood vessel that nourishes the organ – are up to four times more likely to be precipitated.
It offers hope for more accurate screening for cardiovascular disease – the world’s leading cause of death.
Identifying the ‘ticking time bomb’ could save tens of thousands of lives per year.
Heart attacks could be predicted years in advance – via a simple X-ray, according to new research (stock image)
WHAT IS AORTIC CALCULATION?
Aortic valve calcification is a condition in which calcium deposits form on the aortic valve in the heart.
These deposits can cause narrowing when opening the aortic valve.
This narrowing can become severe enough to reduce blood flow through the aortic valve – a condition called aortic valve stenosis.
Factors fueling arteriosclerosis include poor diet, sedentary lifestyle, smoking, and genetics.
Lead author of the study, Professor Josh Lewis, of Edith Cowan University in Perth, Australia, said: “Heart disease is often an assassin.
Many people are not aware that they are at risk or that they have early warning signs, such as calcification of the abdomen or coronary artery.
The abdominal aorta is one of the first places where calcium build-up can occur in the arteries – even before the heart.
‘If we notice this early, we can intervene and make lifestyle and medication changes to help stop the progression of the condition.’
Non-invasive imaging instruments such as CT (computed tomography) scans can produce detailed cross-sectional images of organs and tissues, including the aortic artery.
It can harden when calcium builds up in the wall – and cause cardiac arrest. The condition called AAC (calcification of the abdominal aorta) also causes strokes.
An analysis of data from 52 studies around the world found that the risk of either is two to four times higher.
The study also found the more extensive the mineral, the greater the danger. This was especially true for people with chronic kidney disease.
Non-invasive imaging tools such as CT (computed tomography) scans can produce detailed cross-sectional images of organs and tissues, including the aortic artery (stock image)
Factors that stimulate arteriosclerosis include poor nutrition, a sedentary lifestyle, smoking and genetics.
Professor Lewis hopes the discovery will lead more people to understand their own risk of heart attack or stroke.
He added: “ Calcification of the abdominal aorta is often seen incidentally in many routine tests, such as lateral spine scans from bone density machines or X-rays, and now we have a much better idea of the prognosis in these people when seen.
Factors that promote arteriosclerosis include poor diet, sedentary lifestyle, smoking, and genetics (stock image)
This can provide early warning to doctors to investigate and estimate their patient’s risk of heart attack or stroke.
“Ultimately, if we can identify this condition earlier, people can make lifestyle changes and start preventive treatments earlier, potentially saving many lives in the future.”
The study, published in the Journal of the American Heart Association, builds on his recent research on OC using bone density scans and AI (artificial intelligence).
Dr. Amanda Buttery, of the National Heart Foundation of Australia, said: ‘The researchers found evidence of abdominal aortic calcification in patients with no known cardiovascular disease, suggesting that a more comprehensive cardiovascular risk assessment is required – including blood pressure and cholesterol tests. or a heart defect. Health check.
“The findings are promising and the Heart Foundation would like to see more research in this area.”
Every year in the UK, more than 100,000 people die of a heart attack or related stroke. In the US, the figure is up to eight times higher.
WHY IS NOT THE HEART TIRED?
In a person’s life a human heart can contract billions of times.
The heart is still a muscle, just like the biceps or the hamstring, but the heart never gets tired.
The reason for this rather crucial detail of the anatomy keeps us alive, because without a pumping heart, death will soon follow.
Hearts, although muscles, are made of different fibers than their counterparts.
Also called heart tissue, this type of fiber only exists in the heart and nowhere else in the human body.
The skeletal muscles tire quickly and can switch from aerobic respiration to anaerobic respiration, producing lactic acid that causes cramps.
If this happened in the heart, it would cause a heart attack.
To avoid this and to allow constant use without fatigue, the heart tissue has a different arrangement.
Heart tissue has many more mitochondria that produce a tremendous amount of more energy in the form of a chemical called Adenosine Triphosphate (ATP).
Mitochondria are tiny organelles in cells that are considered the powerhouse of the cell and convert glucose into energy in the organelle.
If you have more of these resources, the heart as an organ will never run out of energy under normal circumstances.
The reason that this regulation does not occur in all muscles is that the energy requirement would be enormous and unsustainable.
The human body would simply demand more energy than it can create.